Genetic requirements for Staphylococcus aureus abscess formation and persistence in host tissues

By Alice G. Cheng, H. Kim, Monica L Burts, T. Krausz, O. Schneewind, D. Missiakas. 2009

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Novel Staphylococcal Glycosyltransferases SdgA and SdgB Mediate Immunogenicity and Protection of Virulence-Associated Cell Wall Proteins (2013, unknown): “These proteins are also thought to be involved in tissue colonization, and elimination of any of them decreases bacterial virulence [5].”
Staphylococcus aureus leukocidins target endothelial DARC to cause lethality in mice (2019, background): “aureus is extravascular (Cheng et al., 2009), the leukocidins are an important strategy for attracting nutrient-rich vascular fluid to the site of infection.”
A Genetic Resource for Rapid and Comprehensive Phenotype Screening of Nonessential Staphylococcus aureus Genes (2013, background): “aureus is ClfA and ClfB dependent but that IsdA, IsdB, Spa, and SdrD are required for kidney abscess formation (2), suggesting that unique factors are required at distinct stages in the pathogenesis process.”
Surface Proteins of Staphylococcus aureus (2019, methodology): “A systematic analysis of mutants defective in individual surface proteins was performed with mutants of strain Newman using a mouse model of bacteremia, dissemination, and abscess formation (102).”
Pathogenicity and virulence of Staphylococcus aureus (2021, background): “insight in abscess formation processes [171].”