Prostanoid receptors: subtypes and signaling.
By R. Breyer, C. Bagdassarian, S. Myers, M. Breyer. Annual Review of Pharmacology and Toxicology, 2001
1,041 citations recorded by the source corpus.
Subfields: Genetics, Pharmacology, Surgery
View Paper: https://www.semanticscholar.org/paper/c5e1785d1e66941220c794cf9a060daadfff212f
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Citations in the corpus
- Urinary bladder contraction and relaxation: physiology and pathophysiology. (2004, background): “In the relaxed smooth muscle cell, Rho is thought to be present in cytosol as Rho-GDP-GDI complexes, and activation in the presence of GTP is catalyzed by Rho-GEF and leads to a translocation of Rho to the cell membrane (237, 614).”
- Eicosanoid actions in insect cellular immune functions (2006, background): “In mammalian systems, the physiological actions of most PGs are mediated by G protein-coupled receptors (GPCRs; Breyer et al., 2001).”
- Pharmacology and signaling of prostaglandin receptors: multiple roles in inflammation and immune modulation. (2004, neutral): “Commonly used selective EP receptor agonists include butaprost (EP2), sulprostone (EP3—also has low affinity for EP1 and FP), and PGE1-OH (EP4; Breyer et al., 2001).”
- GPCR-I-TASSER: A hybrid approach to G protein-coupled receptor structure modeling and the application to the human genome (2015, background): “, 2012), Prostanoids (initiating cancer and inflammation pathways; Breyer et al., 2001), Releasing hormones (progression of cancers; Harrison et al.”