PDE Models for Chemotactic Movements: Parabolic, Hyperbolic and Kinetic
By Benoit Perthame. 2004
154 citations recorded by the source corpus.
Subfields: Biomedical Engineering, Modeling and Simulation, Oncology
View Paper: https://www.semanticscholar.org/paper/be995e9495eb0e54ebdf95637853f56d1c0169ad
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Biomedical Engineering
- José Antonio Carrillo
- Adrien Blanchet
- Nader Masmoudi
- Vincent Calvez
- Nikolaos Bournaveas
- Susana Guti'errez
- R. Kowalczyk
- Zuzanna Szymanska
In Modeling and Simulation
- Nader Masmoudi
- José Antonio Carrillo
- Adrien Blanchet
- Vincent Calvez
- Michael Winkler
- Jacob Bedrossian
- Alexander Lorz
- Zuzanna Szymanska
In Oncology
- Adrien Blanchet
- José Antonio Carrillo
- Nader Masmoudi
- Zuzanna Szymanska
- Vincent Calvez
- Nikolaos Bournaveas
- Susana Guti'errez
- R. Kowalczyk
Citations in the corpus
- Infinite time aggregation for the critical Patlak‐Keller‐Segel model in ℝ2 (2007, methodology): “For further bibliography on the Patlak-Keller-Segel system and related models we refer the interested reader to the surveys [28, 29, 45 ].”
- On the global existence of solutions to an aggregation model (2008, background): “In the literature there are two main classes of models that describe the phenomenon of vasculogenesis: one considers the movement of endothelial cells due to chemotaxis, where a chemical gradient can direct cell migration both up and down a concentration gradient (see [3,4,5]), and the second one re”
- Global Existence for a Kinetic Model of Chemotaxis via Dispersion and Strichartz Estimates (2007, methodology): “The proof starts with the fact that the L1x v-norm of the solution f is a-priori bounded thanks to conservation of mass ∫∫ V f t x v dv dx = ∫∫ V f0 x v dv dx= M (1.2) and then proceeds to bootstrap higher Lpx v-norms based on strong dispersion estimates (see Glassey, 1996; Perthame, 2004a).”