Acute Exacerbation of Idiopathic Pulmonary Fibrosis. An International Working Group Report.
By Harold R Collard, Christopher J. Ryerson, T. Corte, Gisli Jenkins, Y. Kondoh, David J. Lederer, Joyce S Lee, T. Maher, A. Wells, Katerina M. Antoniou and 16 more. American Journal of Respiratory and Critical Care Medicine, 2016
906 citations recorded by the source corpus.
Acute exacerbation of idiopathic pulmonary fibrosis has been defined as an acute, clinically significant, respiratory deterioration of unidentifiable cause. The objective of this international working group report on acute exacerbation of idiopathic pulmonary fibrosis was to provide a comprehensive update on the topic. A literature review was conducted to identify all relevant English text publications and abstracts. Evidence-based updates on the epidemiology, etiology, risk factors, prognosis, and management of acute exacerbations of idiopathic pulmonary fibrosis are provided. Finally, to better reflect the current state of knowledge and improve the feasibility of future research into its etiology and treatment, the working group proposes a new conceptual framework for acute respiratory deterioration in idiopathic pulmonary fibrosis and a revised definition and diagnostic criteria for acute exacerbation of idiopathic pulmonary fibrosis.
Subfields: Physiology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/bc40204fee739bbbcc10147bc2ce9c652fa2ffd5
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In Pulmonary and Respiratory Medicine
Citations in the corpus
- Idiopathic Pulmonary Fibrosis (an Update) and Progressive Pulmonary Fibrosis in Adults: An Official ATS/ERS/JRS/ALAT Clinical Practice Guideline (2022, background): “An additional criterion that was considered by the committee was acute exacerbation, but this was deemed not appropriate for the definition of PPF because it has its own separate definition (132).”
- Diagnosis of Idiopathic Pulmonary Fibrosis. An Official ATS/ERS/JRS/ALAT Clinical Practice Guideline (2018, neutral): “, an unexplained worsening of dyspnea over a few weeks and new ground-glass opacification on HRCT scan with a background of lower lobe fibrotic lung disease) (10).”