Pathophysiology of ischemic acute kidney injury

By A. Sharfuddin, B. Molitoris. Nature Reviews Nephrology, 2011

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Renal ischemia/reperfusion injury; from pathophysiology to treatment (2015, background): “The inevitable injuries may occur after infarction, sepsis and organ transplantation and this phenomena exacerbate tissue damage by initiating an inflammatory cascade including reactive oxygen species (ROS), cytokines, chemokines, and leukocytes activation (1,2).”
Discovery and validation of cell cycle arrest biomarkers in human acute kidney injury (2013, background): “Furthermore, these molecules are known to be associated with mechanisms recently implicated in the pathogenesis of AKI [15-17].”