Sertraline treatment of major depression in patients with acute MI or unstable angina.
By A. Glassman, C. O’Connor, Robert M. Califf, K. Swedberg, P. Schwartz, J. Bigger, K. Krishnan, Louis T. van Zyl, J. Swenson, M. Finkel and 7 more. Journal of the American Medical Association (JAMA), 2002
1,469 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine
View Paper: https://www.semanticscholar.org/paper/ba4103600f2fa34409cbf87d9b02867f09a7c970
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In Cardiology and Cardiovascular Medicine
Citations in the corpus
- Depression as a Risk Factor for Mortality in Patients With Coronary Heart Disease: A Meta-analysis (2004, background): “It is unknown at the moment how best to treat comorbid depressive patients with CHD because of the limited efficacy of psychotherapeutic (71) or psychopharmacologic interventions (72).”
- Depression as a Risk Factor for Coronary Artery Disease: Evidence, Mechanisms, and Treatment (2004, methodology): “The recently completed SADHART trial compared the effects of sertraline and placebo for 24 weeks in MDD patients with unstable angina or recent MI (124).”
- Evidence-based guidelines for treating depressive disorders with antidepressants: A revision of the 2008 British Association for Psychopharmacology guidelines (2015, result): “In patients following an MI or suffering from unstable angina, three SSRI studies with sertraline (Glassman et al., 2002), fluoxetine (Strik et al.”
- Effects of citalopram and interpersonal psychotherapy on depression in patients with coronary artery disease: the Canadian Cardiac Randomized Evaluation of Antidepressant and Psychotherapy Efficacy (CREATE) trial. (2007, background): “While there is a clear need for additional studies evaluating interventions to prevent the cardiac prognostic impact of depression,5,6 there have also been few adequately controlled trials evaluating whether depression treatments are effective in reducing depressive symptoms in patients with CAD.7,8”
- Depression and cardiovascular disease: a clinical review. (2014, background): “There was no reduction in cardiovascular events from treating the depression in the ENRICHD and SADHART studies.(16,17) The CREATE study did not have clinical events as a primary end-point.”