Lack of long-term cortical reorganization after macaque retinal lesions

229 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Frontiers in Computational Neuroscience a Model for Cortical Rewiring following Deafferentation and Focal Stroke (2009, unknown): “…there remains a gap between experimental methods observing plastic connectivity of cell assemblies on the one side and anatomical maps and circuits on the other, it is still unclear to what extent structural plasticity affects anatomical map reorganization (Calford, 2002; Smirnakis et al., 2005).”
A Simple Rule for Dendritic Spine and Axonal Bouton Formation Can Account for Cortical Reorganization after Focal Retinal Lesions (2013, background): “Since axonal growth in the mature cortex is restricted to a few hundreds of micrometers [54], this would further explain why the compensatory capacity of cortical networks is limited and larger LPZs do not recover [87,88].”
Activity-dependent structural plasticity. (2009, background): “…of pre-existing, but latent, horizontal connections (reviewed in Sanes and Donoghue, 2000) and modulation of synaptic efficacy by long-term potentiation (LTP) (Hess and Donoghue, 1994; Hess et al., 1996; but see also Smirnakis et al., 2005) or long-term depression (LTD) (Hess and Donoghue, 1996).”
Tuning out the noise: Limbic-auditory interactions in tinnitus (2010, unknown): “Lesion-induced plasticity in the adult brain has also been documented in the somatosensory and visual systems (Calford et al., 2005; Florence and Kaas, 1995) (but see Smirnakis et al., 2005).”