FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting.
By M. Riminucci, M. Collins, N. Fedarko, Natasha Cherman, A. Corsi, K. White, S. Waguespack, Anurag Gupta, T. Hannon, M. Econs and 2 more. Journal of Clinical Investigation, 2003
621 citations recorded by the source corpus.
Subfields: Nephrology, Oncology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/b2b528e773c88db8c9266f0ce8805ad657bedb9e
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- Q. Luo
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- Shiguang Liu
Citations in the corpus
- Fibroblast growth factor 23 is a counter-regulatory phosphaturic hormone for vitamin D. (2006, background): “Increased production of FGF23 is the cause of phosphaturia and disordered vitamin D metabolism in tumor-induced osteomalacia (6,7), X-linked hypophosphatemic rickets (XLH) (7,8), and McCune-Albright syndrome (9).”