Haemagglutinin mutations responsible for the binding of H5N1 influenza A viruses to human-type receptors
By Shinya Yamada, Yasuo Suzuki, Takashi Suzuki, M. Le, C. Nidom, Yuko Sakai-Tagawa, Y. Muramoto, Mutsumi Ito, Maki Kiso, T. Horimoto and 13 more. Nature, 2006
654 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Infectious Diseases
View Paper: https://www.semanticscholar.org/paper/b0c067fe55ff65b5413c6b0694176142b70be8ee
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Epidemiology
- A. García‐Sastre
- J. Peiris
- M. D. de Jong
- R. Medina
- Yi Guan
- Yong‐Zhen Zhang
- Edward C. Holmes
- D. Morens
In Infectious Diseases
Citations in the corpus
- The Evolution and Emergence of RNA Viruses (2009, background): “2006), whereas analyses of H5N1 viruses have identifi ed a role for changes at HA1 positions 182 and 192 (Yamada et al. 2006).”
- Influenza A viruses: new research developments (2011, background): “, indicating that different residues are responsible for receptor-binding specificity in H5N1 viruse”
- Structural and Functional Bases for Broad-Spectrum Neutralization of Avian and Human Influenza A Viruses (2009, neutral): “Three-dimensional comparisons of the epitope in the five known crystal structure subtypes (three from group 1 (H1, H5 and H9) and two from group 2 (H3 and H7”
- Avian Influenza Virus (H5N1): a Threat to Human Health (2007, background): “On the other hand, some H5N1 viruses isolated from humans appear to have acquired mutations in HA associated with a change in affinity from SA -2,3 and SA -2,6 receptors (263), although such mutations were not by themselves sufficient for these viruses to be efficiently transmitted from human to hum”
- Cross-Species Virus Transmission and the Emergence of New Epidemic Diseases (2008, background): “An analogous process of host adaptation is being suggested for the high-pathogenicity avian H5N1 influenza A virus in various avian hosts, some of which may be gaining mutations associated with mammalian or human adaptation (Table 5) (10, 31, 114, 150).”