Superoxide dismutase multigene family: a comparison of the CuZn-SOD (SOD1), Mn-SOD (SOD2), and EC-SOD (SOD3) gene structures, evolution, and expression.

By I. Zelko, T. Mariani, R. Folz. Free Radical Biology & Medicine, 2002

2,193 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/b0a4b6b932f80a60d48fb5331d1448c11ab0addf

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Citations in the corpus

Free radicals, antioxidants and functional foods: Impact on human health (2010, background): “[45,46] SOD enzymes are present in almost all aerobic cells and in extracellular fluids.”
THE ROLE AND EVOLUTION OF SUPEROXIDE DISMUTASES IN ALGAE 1 (2005, background): “This key antioxidant has been well studied in many eukaryotic systems, including metazoa and plants (Bowler et al., 1992; Scandalios, 1993; Fridovich, 1995; Raychaudhuri and Deng, 2000; Zelko et al., 2002).”
Superoxide Dismutases: Role in Redox Signaling, Vascular Function, and Diseases (2011, neutral): “Regarding other mechanisms of regulation of SODs and their role in diseases, please refer to other reviews (63, 156, 243, 251).”
Measurement and Clinical Significance of Biomarkers of Oxidative Stress in Humans (2017, background): “There are three isoforms of SOD, with a different cellular localization and metal cofactor: homodimeric Cu/Zn-SOD localized in the cytosol and in the mitochondrial intermembrane space, homotetrameric Cu/Zn-SOD with an extracellular distribution, and homotetrameric Mn-SOD localized in the mitochondri”
Regulation of superoxide dismutase genes: implications in disease. (2009, background): “The association between the R213G ECSOD polymorphism and clinical significance has been summarized in several reviews of ECSOD [20,42,183].”
Nrf2 and Oxidative Stress: A General Overview of Mechanisms and Implications in Human Disease (2022, background): “SOD isoenzymes include Cu/Zn-SOD (SOD1), Mn-SOD (SOD2), and extracellular (EC) SOD (SOD3) [31,32].”
Oxidative Stress and Antioxidant Defense (2012, background): “The relatively high EC-SOD level in the lung with its specific binding to the extracellular matrix components may represent a fundamental component of lung matrix protection.(68) H2O2 that is produced by the action of SODs or the action of oxidases, such as xanthine oxidase, is reduced to”
Physiological functions of mineral micronutrients (Cu, Zn, Mn, Fe, Ni, Mo, B, Cl). (2009, background): “Manganese (Mn) Mn 10–100 Mn-superoxide dismutase [51] PEP-carboxykinase [3] Allantoate amidohydrolase [71] Malic enzyme [3] Isocitrate lyase [72] PEP carboxylase [73]”