TGF-β: the master regulator of fibrosis

By Xiao-ming Meng, D. Nikolic-Paterson, H. Lan. Nature Reviews Nephrology, 2016

3,031 citations recorded by the source corpus.

Subfields: Molecular Biology, Nephrology

View Paper: https://www.semanticscholar.org/paper/afa7a16f23be544ebdb9caa304df839c6a3cf77e

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1. Dan-Qian Chen
2. Yan-ni Wang
3. He-He Hu
4. Ying-yong Zhao
5. Gang Cao
6. N. Vaziri
7. Ya-long Feng

Citations in the corpus

• New insights into TGF-β/Smad signaling in tissue fibrosis. (2018, background): “TGF-β1 is expressed in endothelial, hematopoietic, and connective tissue cells, and TGF-β2 is expressed in epithelial and neuronal cells, while TGF-β3 is expressed primarily in mesenchymal cells [1,17,18].”
• Fibroblast-specific TGF-β-Smad2/3 signaling underlies cardiac fibrosis. (2017, neutral): “Global TGF-β activity is upregulated in diseased hearts, where one of its primary actions is directed at the resident fibroblasts to alter the fibrotic response and ventricular remodeling (47, 48).”