Diabetic cardiomyopathy revisited.
By S. Boudina, E. D. Abel. Circulation, 2007
1,589 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/af9c9efada1ef093fca855af45319acc3db6fc16
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In Cardiology and Cardiovascular Medicine
In Molecular Biology
Citations in the corpus
- Diabetic cardiomyopathy, causes and effects (2010, background): “An increase in myocardial fatty acid uptake and oxidation has been described in humans with both Type 1 and Type 2 diabetes, as well as in many animal models [17, 23, 24].”
- Mechanisms of diabetic complications. (2013, background): “this has been termed diabetic cardiomyopathy (53, 509).”
- Mammalian lipoxygenases and their biological relevance (2014, neutral): “cardiomyopathy and other vascular diseases [363].”
- Cardiac Metabolism in Heart Failure - Implications beyond ATP production (2013, neutral): “In diabetic cardiomyopathy, ROS have also been associated with myocardial lipid accumulation, lipotoxicity, and decreased cardiac efficiency on the basis of mitochondrial uncoupling.(98) The failing heart is believed to be subjected to higher levels of free FAs, probably as a result of increased lip”
- Mitochondrial Dysfunction in Diabetes: From Molecular Mechanisms to Functional Significance and Therapeutic Opportunities (2010, neutral): “Metabolic inflexibility may also affect the vascular complications of diabetes, because a switch to glucose utilization represents an important cardiac adaptation to stress (33).”
- Mitochondrial Energetics in the Heart in Obesity-Related Diabetes (2007, background): “In many studies in rodents and humans, there is a characteristic increase in fatty acid utilization and an accompanying reduction in glucose and lactate utilization (1,2).”