It's time for a change to a troponin standard.

By Allan S. Jaffe, J. Ravkilde, R. Roberts, U. Näslund, F. Apple, M. Galvani, H. Katus. Circulation, 2000

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Management of acute coronary syndromes in patients presenting without persistent ST-segment elevation. (2002, background): “Accordingly, the detection of cardiac troponin T and troponin I is specific for myocardial damage, attributing these markers the role of a new gold standard([43]).”
Consideration of a new definition of clinically relevant myocardial infarction after coronary revascularization: An expert consensus document from the society for cardiovascular angiography and interventions (SCAI) (2014, positive): “cTn is a more sensitive and specific biomarker for myonecrosis than is CK-MB (20,21), and CK-MB is no longer measured at some institutions.”
Universal definition of myocardial infarction. (2007, background): “Although elevations in these biomarkers reflect myocardial necrosis, they do not indicate its mechanism.(3,4) Thus, an elevated value of cardiac troponin in the absence of clinical evidence of ischaemia should prompt a search for other aetiologies of myocardial necrosis, such as myocarditis, aortic ”
Biomarkers in acute cardiac disease: the present and the future. (2006, background): “The cTn biomarkers, assuming high-quality analytic methods, whether for cTnI or cTnT, are highly specific and sensitive for cardiac injury ( 12 ).”