Functional neuroimaging and the neuroanatomy of obsessive-compulsive disorder.
By S. Saxena, Scott L. Rauch. Psychiatric Clinics of North America, 2000
857 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Clinical Psychology, Psychiatry and Mental health
View Paper: https://www.semanticscholar.org/paper/ae516c5ea6779138bbea21b1c6fe6ac2a83cbc99
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- Marc N. Potenza
- Samuel R. Chamberlain
- Barbara J. Sahakian
- Naomi A. Fineberg
- E. Hollander
- Susan Nolen-Hoeksema
- Trevor W. Robbins
- David F. Tolin
In Clinical Psychology
- Susan Nolen-Hoeksema
- E. Watkins
- R. Brady
- S. Hannan
- Trevor W. Robbins
- Samuel R. Chamberlain
- Barbara J. Sahakian
- David F. Tolin
In Psychiatry and Mental health
- Trevor W. Robbins
- Samuel R. Chamberlain
- Barbara J. Sahakian
- Naomi A. Fineberg
- Marc N. Potenza
- E. Hollander
- Susan Nolen-Hoeksema
- E. Watkins
Citations in the corpus
- The neuropsychology of obsessive compulsive disorder: the importance of failures in cognitive and behavioural inhibition as candidate endophenotypic markers. (2005, positive): “Given the structural and functional abnormalities in orbitofrontal cortex, anterior cingulate gyrus, and the basal ganglia (especially caudate) (Saxena et al., 2001a,b; Saxena and Rauch, 2000), it is logical to hypothesise that OCD patients would show impaired performance on neurocognitive tasks sub”
- Dysfunctional action monitoring hyperactivates frontal-striatal circuits in obsessive-compulsive disorder: an event-related fMRI study (2005, positive): “…in regions of interest typically associated with action monitoring (caudal and rostral ACC, LPFC) and regions of interest typically associated with OCD (OFC, caudate, thalamus, posterior cingulate; Baxter et al., 1992; Breiter et al., 1996; Rauch et al., 1994, 2001, 2002; Saxena and Rauch, 2000).”
- Obsessional Beliefs and Symptoms of Obsessive–Compulsive Disorder in a Clinical Sample (2008, background): “There is not consensus around the cognitive model, however, and other authors have emphasized behavioral (e.g., Foa and Franklin 2001), information-processing (e.g., McNally 2000; Otto 1992), and neurobiological (e.g., Pitman 1987; Rauch and Savage 2000; Saxena and Rauch 2000 ) mechanisms of OCD.”
- The cognitive neuropsychology of obsessive-compulsive disorder: A critical review (2015, positive): “As a result, individuals with OCD exhibit a bias toward, and excessive attention to, threatening stimuli and may consequently engage in compulsive behaviors (Pauls et al., 2014; Saxena & Rauch, 2000).”
- Should addictive disorders include non-substance-related conditions? (2006, background): “Although both PG and OCD subjects score highly on measures of compulsivity, high scores in PG subjects appear limited to impaired control over mental activities and urges/worries about losing control over motor behaviors [46].”
- Obsessive-Compulsive Disorder and Obsessive-Compulsive Spectrum Disorders: Diagnostic and Dimensional Issues (2007, result): “While there is a balance between these two pathways in healthy individuals, OCD sufferers show preference for the direct pathway, thereby increasing activity in the orbitofrontal cortex, ventromedial caudate, and medial dorsal thalamus, which results in obsessions and compulsions.(60) Although previ”
- A Heuristic for Developing Transdiagnostic Models of Psychopathology (2011, background): “…brain regions implicated in obsessive thoughts and compulsive behaviors (the frontal cortex, basal ganglia, and thalamus; see Rauch et al., 2007; Saxena & Rauch, 2000), in combination with a tendency to ruminate over the meaning of such thoughts and behaviors (Salkovskis, 1999), could…”