A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder

By Amber E. Baum, N. Akula, M. Cabanero, I. Cardona, W. Corona, B. Klemens, T. Schulze, S. Cichon, M. Rietschel, M. Nöthen and 10 more. Molecular Psychiatry, 2008

683 citations recorded by the source corpus.

Subfields: Genetics, Psychiatry and Mental health

View Paper: https://www.semanticscholar.org/paper/acf20b7450166c4bea1ac7776dcd5005db1acc56

How researchers see this paper

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In Genetics

1. Francis J. McMahon
2. Trevor W. Robbins
3. J. Wendland

In Psychiatry and Mental health

1. Francis J. McMahon
2. Trevor W. Robbins
3. J. Wendland
4. J. Dalley
5. Joseph E LeDoux

Citations in the corpus

• The Neuropsychopharmacology of Fronto-Executive Function: Monoaminergic Modulation (2009, background): “A genome-wide association study, which examined more than 550,000 genetic polymorphisms in independent samples, found that mutations in DGKH are most robustly linked to bipolar disorder (Baum et al. 2008), and this finding has now been replicated in a separate, large sample (Wellcome 2007).”
• Genetics of bipolar disorder. (2011, background): “As of early 2010, no single BPD sample has yielded a p value below this threshold in a GWAS (Wellcome Trust Case Control Consortium 2007; Baum et al. 2008; Ferreira et al. 2008; Hattori et al. 2009; Scott et al. 2009; Sklar et al. 2008; Smith et al. 2009).”