IGF-I receptor mutations resulting in intrauterine and postnatal growth retardation.

By M. Abuzzahab, A. Schneider, A. Goddard, F. Grigorescu, C. Lautier, E. Keller, W. Kiess, J. Klammt, J. Kratzsch, D. Osgood and 5 more. New England Journal of Medicine, 2003

623 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Genetics, Pediatrics, Perinatology and Child Health

View Paper: https://www.semanticscholar.org/paper/ab3b0a16b3f6c74e4ced994e2ac62e32f964eb6f

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Citations in the corpus

• Endocrine regulation of human fetal growth: the role of the mother, placenta, and fetus. (2006, background): “sociated with poor prenatal and postnatal growth (139).”
• Genetic and cell biological analysis of integrin outside-in signaling. (2009, background): “Integrin-mediated control of insulin-like growth factor (IGF-1) receptor (IGF1R) signaling IGF-1 and IGF1R play important roles in growth and development; mutations in IGF-1 or IGF1R result in growth retardation in both mice and humans (Liu et al. 1993; Arends et al. 2002; Abuzzahab et al. 2003).”