Th2 and regulatory immune reactions are increased in immunoglobin G4‐related sclerosing pancreatitis and cholangitis

By Y. Zen, T. Fujii, K. Harada, M. Kawano, Kazunori Yamada, M. Takahira, Y. Nakanuma. Hepatology, 2007

671 citations recorded by the source corpus.

Subfields: Hepatology, Rheumatology, Surgery

View Paper: https://www.semanticscholar.org/paper/a9dcbcf0e8d095c1e3513f521b5eda521fd81dce

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Citations in the corpus

• IgG4-Related Disease: A Cross-sectional Study of 114 Cases (2010, neutral): “Head and neck 23 11/12 (48%) 64 (45-74) 725 (29-1420) (n=9)z 9 (39%) Thoracic 16 12/4 (75%)w 59 (43-76) 324 (25-1230) (n=9) 5 (31%) HPB 27 23/4 (85%)w 65 (55-79) 345 (141-760) (n=8) 11 (41%) Retroperitoneal 13 11/2 (83%)w 68 (58-78) 288 (55-559) (n=10) 5 (38%) Systemic 35 30/5 (86%)w 63 (42-77) 1038”
• A novel clinical entity, IgG4-related disease (IgG4RD): general concept and details (2011, neutral): “autoimmune and nonautoimmune diseases [72–74].”
• EASL Clinical Practice Guidelines: management of cholestatic liver diseases. (2009, background): “Preliminary data suggest that immunopathogenesis of IAC strikingly differs from other immunemediated cholestatic liver diseases like PSC and PBC in that T helper 2 (Th2) and T regulatory (Treg) cytokines were markedly overexpressed in IAC patients [158].”