Pathogenesis of liver fibrosis.

By V. Hernández-Gea, S. Friedman. Annual Review of Pathology, 2011

1,073 citations recorded by the source corpus.

Subfields: Cell Biology, Epidemiology, Hepatology

View Paper: https://www.semanticscholar.org/paper/a91a78874d2e71ecafc5b18a1dca27478ec26824

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cell Biology

1. H. Fang
2. R. L. Judd

In Epidemiology

1. Steven Dooley
2. H. Fang
3. R. L. Judd

In Hepatology

1. Steven Dooley
2. H. Fang
3. R. L. Judd

Citations in the corpus

• Adiponectin Regulation and Function (2018, positive): “Activated HSCs are proliferative and play a central role in the development of fibrosis by secreting collagen and other ECMs and releasing chemotactic factors that recruit immune cells to the injury site (100) (Fig.”
• Hepatic Stellate Cells and Liver Fibrosis (2013, neutral): “, sarcoidosis), and drug toxicity, among others (102).”
• TGF-β in Hepatic Stellate Cell Activation and Liver Fibrogenesis—Updated 2019 (2019, background): “Fibrosis means that an excessive amount of fibrillar extracellular matrix (ECM) proteins, e.g., collagen I and III, is deposited in the space of Disse [1,2].”