Plasmid-Mediated Quinolone Resistance: a Multifaceted Threat

By J. Strahilevitz, G. Jacoby, D. Hooper, Ari Robicsek. Clinical Microbiology Reviews, 2009

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Aminoglycoside Modifying Enzymes (2010, background): “N IH -PA Author M anuscript N IH -PA Author M anuscript N IH -PA Author M anuscript qnrB2, qnrB4, qnrB6, qnrB10, qnrS1, qnrS2, and qepA or β-lactamase genes such as blaCTX-M-1, blaCTX-M-14, blaCTX-M-15, blaCTX-M-24, blaDHA-1, blaSHV-12, and blaKPC-2 (Strahilevitz et al., 2009).”
Aminoglycosides: An Overview. (2016, background): “Last, a variant of AAC(60)-Ib that has acquired the ability to modify fluoroquinolones without significantly altering its activity against aminoglycosides has been identified in clinical isolates of Gram-negative bacteria (Robicsek et al. 2006; Strahilevitz et al. 2009).”
Antimicrobial use in aquaculture re-examined: its relevance to antimicrobial resistance and to animal and human health. (2013, background): “…resistance and increase quinolone concentrations required to prevent chromosomal mutations to these antimicrobials, and as a result complicate treatment of infections caused by these pathogens (Oppegaard and Sørum, 1994; Izumi and Aranishi, 2004; Strahilevitz et al., 2009; Shah et al., 2012a,b).”