Eukaryotic polymerases ι and ζ act sequentially to bypass DNA lesions

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Response of human DNA polymerase ι to DNA lesions (2001, background): “(26) and a slight preference for A incorporation opposite the 3′ T of a template TT (6–4) photoproduct was also noticed by Tissier et al.”
A novel function of DNA polymerase ζ regulated by PCNA (2006, background): “Polz efficiently extends mismatched primer termini as well as those containing a terminal nucleotide opposite a noncoding or helix-distorting lesion (Johnson et al, 2000; Guo et al, 2001; Haracska et al, 2001b; Simhadri et al, 2002).”
In Vivo Consequences of Putative Active Site Mutations in Yeast DNA Polymerases α, ε, δ, and ζ (2001, background): “The base substitution specificity of the Johnson et al. 2000), such that loss of this function could explain the observed antimutator effect.”
Regulation of the Rev1–pol ζ complex during bypass of a DNA interstrand cross‐link (2015, unknown): “Pol f is remarkably efficient at extending abnormal primer termini (Johnson et al , 2000; Prakash & Prakash, 2002; Gan et al , 2008).”
Role of DNA Polymerase η in the Bypass of a (6-4) TT Photoproduct (2001, methodology): “pression plasmid pBJ760 (14), generating plasmids pBJ763 and pBJ765, respec-”