Calcium, ischemia and excitotoxicity.
By Kinga Szydłowska, M. Tymianski. Cell Calcium, 2010
748 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/9d2c2efaf80de9a584cd21993e8460f2e7bfa27c
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In Cellular and Molecular Neuroscience
In Molecular Biology
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- Douglas R. Green
- M. Krenz
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- Christopher P. Baines
- Theodore J Kalogeris
- Guido Kroemer
Citations in the corpus
- Noise-induced cochlear neuropathy is selective for fibers with low spontaneous rates. (2013, background): “Studies of glutamate excitotoxicity in central neurons suggest that Ca(2) entry is an important upstream event in the excitotoxicity cascade, and that mitochondria are a crucial source of Ca buffering capacity (Szydlowska and Tymianski 2010).”
- Ischemia/Reperfusion (2016, neutral): “Using the negative ΔΨm created by I/R, this transporter drives movement of the positively charged Ca2+ ions into the mitochondrial matrix (153, 759, 766) (Fig.”
- Cell Death Signaling. (2015, background): “Excitotoxicity is associated with I/R and may occur in other pathological conditions (Szydlowska and Tymianski 2010).”
- Synaptopathy in the noise-exposed and aging cochlea: primary neural degeneration in acquired sensorineural hearing loss (2015, background): “…because, in the central nervous system where it has been best studied, the cell death cascade initiated by glutamate excitotoxicity is thought to involve Ca þþ overload, and one of the most important intracellular Ca þþ buffering systems is the mitochondria (Szydlowska and Tymianski, 2010).”
- Cell Biology of Ischemia/Reperfusion Injury (2012, background): “One of the ways cells deal with this lethal increase in Ca2þ is to take it up into the mitochondria via the mitochondrial Ca2þ uniporter, a protein that uses the negative Dcm to drive uptake of the positively charged Ca2þ ions into the matrix (Contreras et al., 2010; Szydlowska and Tymianski, 2010; ”