SOX9 is required for maintenance of the pancreatic progenitor cell pool

By Philip A. Seymour, K. Freude, M. N. Tran, E. Mayes, J. Jensen, R. Kist, G. Scherer, M. Sander. Proceedings of the National Academy of Sciences of the United States of America, 2007

611 citations recorded by the source corpus.

Subfields: Molecular Biology, Surgery

View Paper: https://www.semanticscholar.org/paper/9adf28d681fe9922bdfeb989e9e28f64139ee82e

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In Molecular Biology

1. C. Wright
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Citations in the corpus

• Pancreatic β-Cell Dedifferentiation As Mechanism Of Diabetic β-Cell Failure (2012, neutral): “FoxO1-Deficient b Cells Revert to an Uncommitted Endocrine Progenitor-like Stage We probed the differentiation stage of FoxO1-deficient, insulindepleted b cells, using markers of pre-endocrine progenitors (Sox9), endocrine progenitors (Neurog3), and committed endocrine cells (ChromograninA, ChgA) (S”
• The Sox Family of Transcription Factors: Versatile Regulators of Stem and Progenitor Cell Fate (2013, neutral): “pancreatic progenitors Seymour et al. (2007);”
• Pancreas organogenesis: From bud to plexus to gland (2011, background): “Sox9 expression persists in the endocrine/duct bipotent progenitor pool located in the central epithelial cords of the maturing epithelial arbor during the secondary transition (Seymour et al., 2007; Lynn et al., 2007).”