Lisinopril-Mediated Regression of Myocardial Fibrosis in Patients With Hypertensive Heart Disease
By C. Brilla, R. Funck, H. Rupp. Circulation, 2000
652 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine
View Paper: https://www.semanticscholar.org/paper/9ad9d93ea79640bbd8499f6bfe69363b4b55f834
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Citations in the corpus
- Calcific aortic stenosis: a disease of the valve and the myocardium. (2012, background): “It is therefore encouraging that in patients with hypertension, ACE inhibitors and angiotensin receptor blockers reduce left ventricular hypertrophy beyond their effects on blood pressure, with favorable effects on myocardial fibrosis, diastolic function, and clinical outcomes (88,89).”
- Myocardial T1 mapping and extracellular volume quantification: a Society for Cardiovascular Magnetic Resonance (SCMR) and CMR Working Group of the European Society of Cardiology consensus statement (2013, neutral): “mechanical [25-27], electrical [28-31], and vasomotor”
- The Pathogenesis of Cardiac Fibrosis (2013, neutral): “Attenuated fibrosis was associated with improved diastolic function [281].”