Loss-of-function variations within the filaggrin gene predispose for atopic dermatitis with allergic sensitizations.

By S. Weidinger, Thomas Illig, H. Baurecht, A. Irvine, E. Rodriguez, A. Díaz‐Lacava, N. Klopp, Stefan Wagenpfeil, Yiwei Zhao, H. Liao and 10 more. Journal of Allergy and Clinical Immunology, 2006

626 citations recorded by the source corpus.

Subfields: Cell Biology, Dermatology, Immunology

View Paper: https://www.semanticscholar.org/paper/982b3849fe554283fcf193ecfc8efadf31ec0182

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Citations in the corpus

• The Atopic March: Progression from Atopic Dermatitis to Allergic Rhinitis and Asthma (2014, background): “AD patients carrying filaggrin mutations are significantly associated with the extrinsic form of the disease (IgE-mediated sensitization to inhalant or food allergens), and the development of allergic rhinitis and asthma [91,103,104].”
• Epidermal barrier dysfunction in atopic dermatitis. (2009, background): “…and found other null mutations within this gene that predispose to AD (Marenholz et al., 2006; Ruether et al., 2006; Sandilands et al., 2006, 2007; Weidinger et al., 2006, 2008; Barker et al., 2007; Hubiche et al., 2007; Morar et al., 2007; Nomura et al., 2007, 2008; Rogers et al., 2007; Stemmler…”