H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failure.

By P. Mishra, N. Tyagi, U. Sen, S. Givvimani, S. Tyagi. 2010

95 citations recorded by the source corpus.

Subfields: Biochemistry, Endocrine and Autonomic Systems, Physiology

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In Physiology

1. R. Khalil

Citations in the corpus

• A Disintegrin and Metalloproteinase (ADAM) and ADAM with Thrombospondin Motifs (ADAMTS) Family in Vascular Biology and Disease (2019, background): “In a mouse model of heart failure induced by aorta-to-vena cava arteriovenous shunt, the cardiac levels of ADAM-12 and tissue fibrosis were increased [163].”
• Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure (2017, unknown): “Several studies have demonstrated that both endogenous and exogenous H2S play key roles in the fibrosis process in many organs, including the heart (104, 108, 132).”