Continued primer synthesis at stalled replication forks contributes to checkpoint activation

By Christopher Van, Shan Yan, W. Michael, S. Waga, Karlene A. Cimprich. 2010

101 citations recorded by the source corpus.

An increased number of primer–template junctions generated by PCNA, Pol-δ, and Pol-ε at stalled replication forks activates Chk1.

Subfields: Cell Biology, Molecular Biology, Oncology

How researchers see this paper

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In Cell Biology

1. Johannes C. Walter

In Molecular Biology

1. Johannes C. Walter
2. Masato T. Kanemaki
3. Sachiko Muramatsu

In Oncology

1. Masato T. Kanemaki

Citations in the corpus

• In silico protein interaction screening uncovers DONSON’s role in replication initiation (2023, background): “S15] and POLA1 [1:5000, (59), used in all POLA1 blots except in Figs.”