Mechanisms of Disease: neuropathic pain—a clinical perspective

By R. Baron. Nature Clinical Practice Neurology, 2006

782 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Neurology, Physiology

View Paper: https://www.semanticscholar.org/paper/8ea7f9d22f585187f7ea8d35d17b6cf3f4ba0bf3

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cognitive Neuroscience

1. T. S. Jensen
2. Rolf-Detlef Treede
3. N. Finnerup
4. R. Baron
5. Niels Birbaumer
6. Clifford J. Woolf
7. Jonathan R. Wolpaw
8. Leonardo G. Cohen

In Neurology

1. Clifford J. Woolf
2. R. Baron
3. T. S. Jensen
4. N. Finnerup
5. Niels Birbaumer
6. N. Attal

In Physiology

1. R. Baron
2. Clifford J. Woolf
3. N. Finnerup
4. T. S. Jensen
5. Rolf-Detlef Treede
6. Niels Birbaumer
7. Ardem Patapoutian
8. N. Attal

Citations in the corpus

• Quantitative sensory testing in the German Research Network on Neuropathic Pain (DFNS): Somatosensory abnormalities in 1236 patients with different neuropathic pain syndromes (2010, background): “nerve lesion induced by virus infection or by diabetes), is the best approach or whether it might be preferable to classify neuropathic pain conditions on the basis of symptoms and signs [27,46,50] or of patterns of somatosensory abnormalities and the likely underlying mechanisms [5,6,12,54].”
• The diagnostic criteria for small fibre neuropathy: from symptoms to neuropathology (2008, background): “The features of neuropathic pain reflected the predominant impairment of Ad and C-fibre, since spontaneous pain, thermal allodynia and hyperalgesia were most frequently reported (Baron, 2006).”
• Prevalence of chronic pain with neuropathic characteristics in the general population (2008, neutral): “This may be related to the specificities of its pathophysiological mechanisms (Woolf and Mannion, 1999; Baron, 2006), but also to its being underestimated, particularly in patients having no definite neurological condition (Harden and Cohen, 2003).”
• Transient receptor potential channels: targeting pain at the source (2009, background): “Following peripheral nerve damage, nociceptors may begin to fire ectopically, and in this way contribute to the spontaneous component of neuropathic pain”
• EFNS guidelines on the pharmacological treatment of neuropathic pain: 2010 revision (2010, background): “The management of NP is challenging because the response to most drugs remains unpredictable [4] despite attempts to develop a more rationale therapeutic approach [5,6].”
• The evidence for pharmacological treatment of neuropathic pain (2010, background): “Such classification is in line with strategies directed at pain mechanisms, which are currently being explored [11,33,119].”