Pathogenetic mechanisms in radiation fibrosis.

By J. Yarnold, M. C. Brotons. Radiotherapy and Oncology, 2010

600 citations recorded by the source corpus.

Subfields: Immunology, Otorhinolaryngology, Radiology, Nuclear Medicine and Imaging

View Paper: https://www.semanticscholar.org/paper/8e51ab246276a4268ad7a5ce3e9cb7ce7bfecdf5

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In Otorhinolaryngology

1. J. Bourhis

Citations in the corpus

• Ultrahigh dose-rate FLASH irradiation increases the differential response between normal and tumor tissue in mice (2014, methodology): “To test this model, 24 hours before radiation,micewere exposed to tumor necrosis factor–a (TNF-a), a key cytokine involved in endothelial cell apoptosis, inflammation, myofibroblast transdifferentiation, and the pathogenesis of radiation pneumonitis (14, 15).”