Light-dependent herbicides: an overview.

By F. Hess. 2000

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Plant Response to Combinations of Mesotrione and Photosystem II Inhibitors1 (2006, background): “The carotenoid system becomes completely overloaded and cannot quench this reactive oxidizing energy (Hess 2000).”
A waterhemp (Amaranthus tuberculatus) biotype with multiple resistance across three herbicide sites of action (2005, background): “The ultimate result of plant death associated with paraquat treatment of susceptible plants is the increase in reactive oxygen species (ROS), resulting in loss of membrane integrity through lipid peroxidation, similar to the phytotoxic effects exerted by inhibitors of PPO or PSII (Hess 2000).”
Reactive oxygen species in plants: their generation, signal transduction, and scavenging mechanisms (2011, background): “This ROS initiates LP of PUFAs (Hess, 2000).”
Chlorophyll fluorescence as a marker for herbicide mechanisms of action (2012, background): “Chlorophyll fluorescence is affected by other light-dependent herbicides, such as the inhibitors of glutamine synthetase, protoporphyrinogen oxidase, and carotenoid biosynthesis [15].”