Pathogenetic mechanisms in thyroid follicular-cell neoplasia
By T. Kondo, S. Ezzat, S. Asa. Nature Reviews. Cancer, 2006
929 citations recorded by the source corpus.
Subfields: Cell Biology, Endocrinology, Diabetes and Metabolism, Oncology
View Paper: https://www.semanticscholar.org/paper/8c0a8ee92c38ec616354d027bcb8430afe054cc0
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In Cell Biology
In Endocrinology, Diabetes and Metabolism
In Oncology
Citations in the corpus
- MicroRNA expression profiling of thyroid tumors: biological significance and diagnostic utility. (2008, background): “Significant information has been accumulated on carcinogenic mutations in thyroid cancer (18).”
- Involvement of members of the cadherin superfamily in cancer. (2009, background): “Whether RET can be considered a cell adhesion receptor is undetermined, but its involvement in cancer is beyond doubt (reviewed by Kondo et al. 2006; Zbuk and Eng 2007).”
- American Thyroid Association Guidelines for Management of Patients with Anaplastic Thyroid Cancer (2012, positive): “Other mutations, such as BRAF and RAS, are common in both well-differentiated thyroid cancer and ATCs and are likely to be early events in thyroid carcinogenesis that may predispose to tumor dedifferentiation (60,81).”
- Variation in cancer risk among tissues can be explained by the number of stem cell divisions (2015, background): “Only ~ 3% of these cancers are derived from parafollicular cells (C cells) (106-108), which are the cell of origin for medullary carcinomas.”