Papillomaviruses Causing Cancer: Evasion From Host-Cell Control in Early Events in Carcinogenesis
By H. Hausen. 2000
1,360 citations recorded by the source corpus.
Subfields: Epidemiology, Periodontics, Surgery
View Paper: https://www.semanticscholar.org/paper/8b0f4d7d50d0c018ffedbc343ee838a4bdbb148d
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Citations in the corpus
- The 2001 Bethesda System: terminology for reporting results of cervical cytology. (2002, methodology): “The dichotomous division of SIL reflects the substantial virological, molecular, and clinical evidence that LSIL is generally a transient infection with HPV, while HSIL is more often associated with viral persistence and higher risk for progression.(10-13) In addition, data from the ASCUS LSIL Triag”
- The epidemiology of genital human papillomavirus infection. (2006, background): “More than 120 different human papillomavirus (HPV) ypes have been catalogued so far [1,2], of which more than 0 infect the epithelial lining of the anogenital tract and other ucosal areas of the body.”
- Papillomaviruses and cancer: from basic studies to clinical application (2002, background): “Infection is commonly transmitted by sexual contact and results initially in inconspicuous squamous intraepithelial lesions (SIL) in women (reviewed in Ref”