Signaling of transforming growth factor‐β family members through Smad proteins
By S. Itoh, F. Itoh, M. Goumans, P. ten Dijke. 2000
536 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/86d16de6c20466600d1a401c20a7745b2220ef72
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Molecular Biology
- R. Derynck
- D. Medici
- David M. Gonzalez
- Nikolaos G. Frangogiannis
- M. Karin
- Wan-Wan Lin
- Xin-Hua Feng
- J. Massagué
Citations in the corpus
- Smad-dependent and Smad-independent pathways in TGF-β family signalling (2003, background): “1 ) is a linear signalling pathway from the type II to the type I receptor kinase to Smad activation, resulting in ligand-induced transcriptio”
- Signaling mechanisms of the epithelial-mesenchymal transition (2014, background): “Both ATF-2 (activating transcription factor-2, a substrate of p38 MAPK pathways) and c-Jun (a substrate of JNK) are capable of interacting with SMADs (80, 81, 89).”