Long-distance signalling and a mutational analysis of branching in pea

By C. Beveridge. Plant growth regulation (Print), 2000

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Carotenoid cleavage dioxygenases and their apocarotenoid products in plants (2009, background): “When the max and rms mutants are grafted to wild-type root stock, the branching phenotype of these mutants is restored, which indicates that their branching is also controlled by long-distance signaling from the root (Beveridge 2000; Morris et al. 2001; Booker et al. 2005; Foo et al. 2005).”
MAX1 and MAX2 control shoot lateral branching in Arabidopsis. (2002, background): “Conversely, the mutations at the RMS branching loci in pea differ in their effects on leaf senescence (Beveridge, 2000), which indicates that delayed leaf senesce is not in general a consequence of increased axillary branching.”
MAX4 and RMS1 are orthologous dioxygenase-like genes that regulate shoot branching in Arabidopsis and pea. (2003, background): “This mobile signal is unlikely to be auxin or cytokinin because, as well as increased branching, the rms1 and rms5 mutants have reduced root-derived cytokinin and have at least WT auxin levels and transport (Beveridge et al. 1997; Morris et al. 2001).”
The strigolactone story. (2010, background): “Therefore, although both auxin and cytokinin appear to play important roles in regulation of shoot branching, the involvement of another signaling molecule, a long distance second messenger of auxin, has been postulated (16, 20).”