Pathophysiology of the basal ganglia in Parkinson's disease.
By Jose A. Obeso, M. Rodríguez-Oroz, Manuel Rodríguez, J. Lanciego, Julio Artieda, N. Gonzalo, C. Olanow, Jos A Obesg, M. C, Neurobiology Laboratoly. Trends in Neurosciences, 2000
859 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Neurology
View Paper: https://www.semanticscholar.org/paper/84d5f00e4d27d8d405d365a7b6f791ddbc5e0465
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In Cellular and Molecular Neuroscience
In Neurology
Citations in the corpus
- Functional neuroanatomy of the basal ganglia. (2012, neutral): “Increased output from the GPi/SNr overinhibits the thalamocortical projection, reducing cortical neuronal activation associated with movement initiation (DeLong 1990; Obeso et al. 2000).”
- Animal models of Parkinson's disease: a source of novel treatments and clues to the cause of the disease (2011, background): “…has been the most instrumental in the search for the cause of treatment-related dyskinesia by allowing electrophysiological, anatomical and biochemical analysis of the alterations occurring in the basal ganglia (see as examples Crossman, 1987; 1989; 1990; 2000; Obeso et al., 2000a,b,c; 2002; 2008).”
- The Basal Ganglia and Cortex Implement Optimal Decision Making Between Alternative Actions (2007, background): “A second type of dopamine release provides tonic or background levels, severe lowering of which can result in Parkinson’s disease (Obeso et al., 2000).”
- Functional organization of the basal ganglia: Therapeutic implications for Parkinson's disease (2008, background): “The BG are currently seen as a highly organized network, where different parts are activated depending on specific functions and circumstances.(52,53) As noted earlier, present evidence suggests that the BG are not only involved in movement control, but also in functions such as learning, planning, ”