Inflammatory processes in renal fibrosis
By Xiao-ming Meng, D. Nikolic-Paterson, H. Lan. Nature Reviews Nephrology, 2014
654 citations recorded by the source corpus.
Subfields: Nephrology, Surgery
View Paper: https://www.semanticscholar.org/paper/83ddc0db88f3fb8c725747522081490e8f3d62a8
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In Nephrology
Citations in the corpus
- New insights into TGF-β/Smad signaling in tissue fibrosis. (2018, background): “chemotactic factors and pro-inflammatory factors in mesangial and inflammatory cells [28,29].”
- TGF-β/Smad signaling in renal fibrosis (2015, background): “…that myofibroblasts may be derived from the resident fibroblasts, pericytes, bone marrow cells (e.g., fibrocytes), epithelial cells (Epithelial–mesenchymal transition, EMT), and endothelial cells (Endothelial–mesenchymal transition, EndMT) (Allison, 2013; LeBleu et al., 2013; Meng et al., 2014).”