Ageing and neuronal vulnerability

By Mark P. Mattson, T. Magnus. Nature Reviews Neuroscience, 2006

1,073 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/83442ac1ee63429d6608cc3edf688ccbd6596949

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. Chunyan Guo
2. Li Sun
3. Xueping Chen
4. Dan-shen Zhang
5. Guido Kroemer
6. Mark P. Mattson
7. B. Barres
8. M. Sofroniew

In Molecular Biology

1. Guido Kroemer
2. P. Golstein
3. S. A. Liddelow
4. Mark P. Mattson
5. B. Barres
6. L. Galluzzi
7. Douglas R. Green
8. Beth Levine

In Physiology

1. Guido Kroemer
2. Chunyan Guo
3. Xueping Chen
4. Li Sun
5. Dan-shen Zhang
6. S. A. Liddelow
7. Mark P. Mattson
8. B. Barres

Citations in the corpus

• Exercise, oxidative stress and hormesis. (2008, neutral): “…a significant role by which exercise decreases the incidence of ROS-associated diseases, including specific cardiovascular diseases, stroke, Alzheimer disease, and certain types of cancer (Perry et al., 2005; Radak et al., 2005; Mattson and Magnus, 2006; Mattson and Wan, 2005; Yu and Chung, 2006).”
• Oxidative stress, mitochondrial damage and neurodegenerative diseases (2013, background): “Mitochondria are also capable of sequestering Ca 2+ and then releasing these ions into the cytosol [31] .”
• Cell death by necrosis: towards a molecular definition. (2007, background): “For example, mouse strains vary in their susceptibility to brain ischemia, and important differences are dictated by the age of the mice and by distinct brain regions [ 17 ].”
• Cellular Stress Responses, The Hormesis Paradigm, and Vitagenes: Novel Targets for Therapeutic Intervention in Neurodegenerative Disorders (2010, neutral): “Oligomers of A b , a -synuclein, and HTT might damage and kill neurons by inducing membrane-associated oxidative stress (MAOS), thereby impairing mitochondrial function and thus causing degenerative cell death (287).”
• Normal aging induces A1-like astrocyte reactivity (2018, background): “Taken together, the impaired mitochondrial function and antioxidant capacity of aged astrocytes could contribute to both metabolic and oxidative stress known to occur in the aged brain (27, 28).”
• Calcium and neurodegeneration (2007, background): “…al ., 2001; Drew & Leeuwenburgh, Calcium and neurodegeneration, M. P. Mattson © Blackwell Publishing Ltd/Anatomical Society of Great Britain and Ireland 2007 No claim to original US government works 339 & Ischiropoulos, 2005; Saito et al ., 2005; Abou-Sleiman et al ., 2006; Mattson & Magnus, 2006).”