Critical Role of ADAMTS2 (A Disintegrin and Metalloproteinase With Thrombospondin Motifs 2) in Cardiac Hypertrophy Induced by Pressure Overload
By Xiaodi Wang, Wen Chen, Jie Zhang, Aiman Khan, Liangpeng Li, F. Huang, Z. Qiu, Liming Wang, Xin Chen. 2017
41 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology, Pathology and Forensic Medicine
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cardiology and Cardiovascular Medicine
In Molecular Biology
In Pathology and Forensic Medicine
Citations in the corpus
- A Disintegrin and Metalloproteinase (ADAM) and ADAM with Thrombospondin Motifs (ADAMTS) Family in Vascular Biology and Disease (2019, background): “ADAMTS-2 knockout mice exhibit severe cardiac hypertrophy and heart failure likely because in the absence of ADAMTS-2 collagen fibrils are abnormal and do not provide the required mechanical strength to counterbalance the pressure overload, and overexpression of ADAMTS-2 in mice reverses this damagi”