Myocardial infarction induces early increased remote ADAM8 expression of rat hearts after cardiac arrest

By Vilma Vuohelainen, E. Raitoharju, Mari Levula, T. Lehtimaki, M. Pelto-huikko, T. Honkanen, A. Huovila, T. Paavonen, M. Tarkka, A. Mennander. 2011

20 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Immunology and Allergy, Molecular Biology

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In Cardiology and Cardiovascular Medicine

1. S. Zhong
2. R. Khalil

In Immunology and Allergy

1. S. Zhong
2. R. Khalil

In Molecular Biology

1. S. Zhong
2. R. Khalil

Citations in the corpus

• A Disintegrin and Metalloproteinase (ADAM) and ADAM with Thrombospondin Motifs (ADAMTS) Family in Vascular Biology and Disease (2019, background): “Although ADAM-8−/− mice develop and grow normally without a disease phenotype [63], ADAM-8 could contribute to cardiac inflammation as its levels are increased in the periinfarct myocardium following myocardial infarction, which is often associated with infiltration of inflammatory cells [64].”