Seminars in cell & developmental biology.
By A. Schindeler. Seminars in Cell and Developmental Biology, 2008
1,041 citations recorded by the source corpus.
Subfields: Biomedical Engineering
View Paper: https://www.semanticscholar.org/paper/75fe0371750c823415c7ee1774db5840882d5770
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In Biomedical Engineering
Citations in the corpus
- The Roles of Autophagy in Cancer (2018, background): “mitochondria induces excessive ROS production, resulting in promotion of carcinogenesis [68,69,70].”
- The interaction between ferroptosis and lipid metabolism in cancer (2020, background): “Many enzymes, including LOXs, NADPH oxidases and cyclooxygenases (COXs) promote endogenous ROS production in cancer.(24) However, not all ROS induce ferroptosis, rather lipid peroxidation is the primary cause of ferroptosis, which was confirmed by lipophilic antioxidants.”
- Oxidative Stress in Cancer (2020, background): “The increase in ROS in cancer cells stimulates proliferation and cell survival by blunting the activities of PTPs, PTEN (Figure 3), and MAPK phosphatases, thereby augmenting MAPKERK, PI3K-Akt, and PKD-NF-κB signaling cascades in a cell-specific manner (Moloney and Cotter, 2018).”
- Targeting cell death pathways for cancer therapy: recent developments in necroptosis, pyroptosis, ferroptosis, and cuproptosis research (2022, background): “Furthermore, excessive proliferation of tumor cells is always accompanied by high ROS production, but these cells optimize ROS-driven proliferation via their effective antioxidant activity, enabling them to adapt and thrive under highly oxidative conditions while preventing ROS from reaching that th”
- ROS-induced lipid peroxidation modulates cell death outcome: mechanisms behind apoptosis, autophagy, and ferroptosis (2023, background): “The general endogenous antioxidant system includes enzyme antioxidants, such as superoxide dismutase, catalases, glutathione peroxidase (GPX), glutathione (GSH) reductase, peroxiredoxin, and thioredoxin (Trx); and non-enzymatic antioxidants, including GSH, vitamins or their analogues (vitamins A, C,”