EFFECTS OF VISUAL DEPRIVATION ON MORPHOLOGY AND PHYSIOLOGY OF CELLS IN THE CATS LATERAL GENICULATE BODY.
By T. Wiesel, David H. Hubel. 1963
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- Alessandro Achille
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- Laurent Itti
- Tommaso Furlanello
- Wenguan Wang
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- Maxim Bazhenov
Citations in the corpus
- Critical Learning Periods in Deep Neural Networks (2017, background): “, retina, lateral geniculate nucleus) are little (or not at all) affected by deficits (4, 16, 46).”
- Active Long Term Memory Networks (2016, background): “To prevent the interference of new experience with previously stored functions, the N networks’s learning rate is drastically reduced during post-developmental phases in an imitation of the visuo-cortical critical period of plasticity [50].”
- Expressive Therapy with Severely Maltreated Children: Neuroscience Contributions (2005, background): “When researchers subsequently looked at the kitten’s brain to study the area that governs sight, it was found to be underdeveloped (Baer & Rittenhouse, 1999; Wiesel & Hubel, 1963, 1965).”
- Heterosynaptic Plasticity Prevents Runaway Synaptic Dynamics (2013, background): “Potentiation or depression of synapses toward extreme weights may be useful for processes requiring strong synaptic competition including elimination of the “wrong” connections, such as segregation of inputs from two eyes during development of the visual cortex (Wiesel and Hubel, 1963; Thompson et a”
- Overcoming Catastrophic Forgetting in Convolutional Neural Networks by Selective Network Augmentation (2018, unknown): “This network was trained during a development phase in a homogeneous environment and to prevent the interference of new task, the learning rate was reduced drastically as an imitation of the visual-cortical critical period of plasticity [15].”
- The period of susceptibility to the physiological effects of unilateral eye closure in kittens (1970, background): “(From Wiesel & Hubel, 1963 b.) u 20 20 10 z 1 2 3 4 5 6 7 Ocular dominance N I I | I I 0 1 2 3 4 5 6 7 8 9 10 Months 30 C $ 20 I.”
- Exploring the Thalamus and its Role in Cortical Function, S.M. Sherman, R.W. Guillery (Eds.). The MIT Press (2006), ISBN: 0-262-19532-1 (2007, background): “…but its reaction to varying, complex regimes of visual deprivation has taught us a great deal about the plasticity and development of thalamocortical connections (Wiesel & Hubel, 1963; Sherman & Spear, 1982; Shatz, 1994; Rittenhouse et al., 1999; Berardi et al., 2003; Heynen et al., 2003).”
- Neuronal Projections from V1 to V2 in Amblyopia (2012, unknown): “In striate cortex, few neurons could be stimulated effectively through the amblyopic eye (Wiesel and Hubel, 1963b; Hubel and Wiesel, 1970).”
- Too many cooks? Intrinsic and synaptic homeostatic mechanisms in cortical circuit refinement. (2011, methodology): “…in refining cortical function, and much work has gone into determining the cellular plasticity mechanisms that underlie various phases of cortical circuit refinement, using a venerable sensory deprivation paradigm pioneered by Hubel and Wiesel decades ago (Hubel & Wiesel 1970, Wiesel & Hubel 1963).”
- From synapse to nucleus: calcium-dependent gene transcription in the control of synapse development and function. (2008, background): “Sensory blockade by monocular deprivation significantly alters dendritic development in both the lateral geniculate nucleus and the visual cortex (Coleman and Riesen, 1968; Wiesel and Hubel, 1963).”
- Spike Timing Dependent Plasticity: A Consequence of More Fundamental Learning Rules (2010, unknown): “Based on observations of the development and plasticity of visual cortex (Wiesel and Hubel, 1963), Bienenstock, Cooper, and Munro (BCM) theorized (Bienenstock et al., 1982) and several groups (Dudek and Bear, 1992; Mulkey and Malenka, 1992) demonstrated experimentally that if a presynaptic neuron…”
- Extent of recovery from the effects of visual deprivation in kittens. (1965, methodology): “(As discussed previously (6), cell sizes vary to some extent from one animal to the next, partly, no doubt, because of differences in fixation.”