Analysis of mitochondrial function in situ in permeabilized muscle fibers, tissues and cells

By A. Kuznetsov, V. Veksler, F. Gellerich, V. Saks, R. Margreiter, W. Kunz. Nature Protocols, 2008

811 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Platelets release mitochondria serving as substrate for bactericidal group IIA-secreted phospholipase A2 to promote inflammation. (2014, methodology): “Unless mitochondria have been isolated and free in the milieu, permeabilization of the cytoplasmic membrane is necessary to allow the access of exogenous substrates added exogenously to stimulate mitochondrial respiration.(27,28) To evaluate respiration by putative mitoMPs in platelet-free supernata”
CD38 dictates age-related NAD decline and mitochondrial dysfunction through a SIRT3-dependent mechanism (2016, neutral): “Citrate synthase was measured as described (Kuznetsov et al., 2008).”