CXCR3 Expression and Activation of Eosinophils: Role of IFN-γ-Inducible Protein-10 and Monokine Induced by IFN-γ1
By Jinquan Tan, C. Jing, H. Jacobi, C. Reimert, A. Millner, S. Quan, J. B. Hansen, S. Dissing, H. Malling, P. Skov and 1 more. Journal of Immunology, 2000
166 citations recorded by the source corpus.
Subfields: Immunology, Oncology, Physiology
View Paper: https://www.semanticscholar.org/paper/6f8313d1e5f52c15cdb470843feccd0a80c6b28f
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Citations in the corpus
- IFN-γ-Inducible Protein 10 (IP-10; CXCL10)-Deficient Mice Reveal a Role for IP-10 in Effector T Cell Generation and Trafficking1 (2002, background): “cells (24, 25), eosinophils (26), dividing microvascular endothelial”
- CXCL10/IP-10 in Infectious Diseases Pathogenesis and Potential Therapeutic Implications (2011, unknown): “p38/MAPK and PI3K signaling play important roles in the CXCL10/CXCR3 chemokine receptor-induced chemotaxis in human airway epithelial cells [36,37,48]; in human type II pneumocytes, CXCL10/CXCR3 interaction induces chemotaxis via mitogen-activated protein kinase (MAPK) and PI 3-kinase pathways (PI3K”