Diminished Pupillary Light Reflex at High Irradiances in Melanopsin-Knockout Mice
By Farhad Abtahian, Anastasia Guerriero, Eric Sebzda, Min Lu, Rong Zhou, A. Mócsai, Erin E. Myers, Bin Huang, D. G. Jackson, Victor A. Ferrari and 5 more. Science, 2003
840 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/6eb3d3206e8a2f1e88e5ef3e3cd34712fdccbffc
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In Cellular and Molecular Neuroscience
In Endocrine and Autonomic Systems
In Molecular Biology
Citations in the corpus
- Melanopsin and rod–cone photoreceptive systems account for all major accessory visual functions in mice (2003, background): “On ablation of the melanopsin gene, retinal ganglion cells that normally express melanopsin are no longer intrinsically photosensitiv”
- A clockwork web: circadian timing in brain and periphery, in health and disease (2003, background): “, and in the absence of melanopsin the 'circadian' RGCs lose their intrinsic photosensitivit”
- Melanopsin-expressing retinal ganglion-cell photoreceptors: cellular diversity and role in pattern vision (2010, background): “The ipRGCs mediate a variety of physiological responses to ambient light, such as circadian photoentrainment and the pupillary light reflex (Guler et al., 2008; Hatori et al., 2008; Lucas et al., 2003; Panda et al., 2002; Ruby et al., 2002).”
- Central projections of melanopsin‐expressing retinal ganglion cells in the mouse (2006, background): “All photic responses to which mRGCs are known to contribute persist in some form when the melanopsin gene is knocked out (Panda et al., 2002; Ruby et al., 2002; Lucas et al., 2003; Mrosovsky and Hattar, 2003).”