From NAFLD to NASH to cirrhosis—new insights into disease mechanisms
By A. Wree, L. Broderick, Ali Canbay, H. Hoffman, A. Feldstein. Nature Reviews Gastroenterology &Hepatology, 2013
605 citations recorded by the source corpus.
Subfields: Cell Biology, Endocrinology, Diabetes and Metabolism, Epidemiology
View Paper: https://www.semanticscholar.org/paper/6dead985e5caf8100a635febb4a059b328e06950
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In Cell Biology
In Endocrinology, Diabetes and Metabolism
In Epidemiology
Citations in the corpus
- NAFLD and diabetes mellitus (2017, background): “SOCS1 and SOCS3 impair insulin signalling through ubiquitin-dependent degradation of IRS (3). c-Jun N-terminal kinase (JNK, also known as mitogen activated protein kinase) represents another important inhibitory kinase of IRS that is activated in response to a variety of extracellular stimuli and ce”