Inflammation and tumor microenvironments: defining the migratory itinerary of mesenchymal stem cells

By Erika L Spaeth, A. Klopp, J. Dembinski, Michael Andreeff, F. Marini. Gene Therapy, 2008

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Mesenchymal Stem Cell Transition to Tumor-Associated Fibroblasts Contributes to Fibrovascular Network Expansion and Tumor Progression (2009, background): “The tropism of MSC for tumors is thought to be due to similarity in factors secreted by wounds and tumors [13], substantiating the concept that tumors are ‘‘wounds that never heal’’ [14,15].”
Accessories to the crime: functions of cells recruited to the tumor microenvironment. (2012, unknown): “…Psaila and Lyden, 2009), and (2) the myofibroblastic/MSC subtype of CAFs, likely also present in metastatic vascular stem cell niches (Kidd et al., 2009; Korkaya et al., 2011; Liu et al., 2011; Spaeth et al., 2008, 2009); similar niche-forming cells may also nurture CSCs inside primary tumors.”