ADAM-mediated ectodomain shedding of HB-EGF in receptor cross-talk.

By S. Higashiyama, D. Nanba. 2005

167 citations recorded by the source corpus.

Subfields: Immunology and Allergy, Oncology

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In Oncology

1. Carlos Fernandez-Patron
2. Pu Zhang
3. Mengcheng Shen
4. Z. Kassiri

Citations in the corpus

• ADAMs family and relatives in cardiovascular physiology and pathology. (2016, background): “Activation of Epidermal Growth Factor Receptor (EGFR), through shedding of membrane-anchored proTGFα [91] and proHB-EGF [92], has been identified among critical functions of ADAM17 [93–96], compared to other candidate HB-EGF sheddases (ADAM9, ADAM12 and ADAM15) [10,97].”