Progesterone receptor contains a proline-rich motif that directly interacts with SH3 domains and activates c-Src family tyrosine kinases.

By Viroj Boonyaratanakornkit, M. Scott, V. Ribon, Lori Sherman, S. Anderson, J. Maller, W. Miller, D. Edwards. Molecules and Cells, 2001

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Overview of Nomenclature of Nuclear Receptors (2006, background): “However, it now seems that at least some of the reported effects can be attributed to the same steroid receptors that are known as NRs (Migliaccio et al., 1996; Simoncini et al., 2000; Boonyaratanakornkit et al., 2001; Castoria et al., 2001; Kousteni et al., 2001).”
Src family kinases in tumor progression and metastasis (2003, background): “[95] demonstrated that the progesterone receptor was capable of directly activating SFKs, through displacement of the SH3 domain/linker intramolecular interaction, in response to progestin.”
Nongenomic actions of steroid hormones (2003, neutral): “uk/interpro/IEntry?ac=IPR001452">SH3 ) domains of various proteins in response to progesteron”
Ovarian follicle growth, maturation, and ovulation in teleost fish (2004, background): “It is tempting to speculate that both novel (Thomas et al. 2002) and classical (Bagovski et al. 2001; Boonyaratanakornkit et al. 2001) forms of progestin receptors are involved in the regulation of MIH-dependent meiotic resumption.”