Cell death and cell death responses in liver disease: mechanisms and clinical relevance.
By Tom Luedde, N. Kaplowitz, Robert F. Schwabe. Gastroenterology, 2014
706 citations recorded by the source corpus.
Subfields: Epidemiology, Hepatology
View Paper: https://www.semanticscholar.org/paper/619ff4e75ce7b4b67614de85d6acd7f8dacfcc97
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Epidemiology
- Robert F. Schwabe
- S. Friedman
- Natascha Roehlen
- E. Crouchet
- T. Baumert
- E. Seki
- Yujin Hoshida
- T. Higashi
In Hepatology
- T. Baumert
- Natascha Roehlen
- E. Crouchet
- E. Seki
- Robert F. Schwabe
- Yujin Hoshida
- S. Friedman
- T. Higashi
Citations in the corpus
- Hepatic stellate cells as key target in liver fibrosis (2017, background): “autophagic cell death), trigger cell death responses [39].”
- Liver Fibrosis: Mechanistic Concepts and Therapeutic Perspectives (2020, background): “As in chronic hepatitis C, chronic hepatitis B triggers HSCs activation via DAMPs release and the host antiviral immune response leading to chronic inflammation [31,35,76,77,192].”
- Hepatic Inflammation and Fibrosis: Functional Links and Key Pathways (2015, background): “Biomarkers of cell death correlate well with risk of fibrosis development.(11) Wound-healing responses to acute injury are beneficial in several ways: (1) Fibrosis provides mechanical stability; (2) inflammatory cells contribute to the removal of cellular debris; and (3) inflammatory signals also ex”