Angiopoietin-2 displays VEGF-dependent modulation of capillary structure and endothelial cell survival in vivo
By I. Lobov, P. Brooks, R. Lang. Proceedings of the National Academy of Sciences of the United States of America, 2002
730 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Oncology
View Paper: https://www.semanticscholar.org/paper/60029f2ec9a0f8c30ad5b2536f206fc2cbab3666
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In Cellular and Molecular Neuroscience
In Molecular Biology
- K. Alitalo
- Gerhard Christofori
- Ernesta Fagiani
- Gavin Thurston
- H. Augustin
- G. Koh
- Rakesh K. Jain
- Peter Carmeliet
In Oncology
Citations in the corpus
- Angiopoietins in angiogenesis. (2013, background): “The EC of unstable vessels lacking pericytes have different fates: they die in the absence of VEGF, but migrate or proliferate in the presence of VEGF depending on whether they are located at the sprouting tip or the stalk of a newly forming blood vessel [93].”
- Control of vascular morphogenesis and homeostasis through the angiopoietin–Tie system (2009, background): “In the presence of VEGF, ANG2 enables EC migration, proliferation and the sprouting of new blood vessels, whereas the presence of ANG2 leads to EC death and vessel regression if the activity of endogenous VEGF is inhibite”