Nonalcoholic fatty liver, steatohepatitis, and the metabolic syndrome

By Giulio Marchesini, E. Bugianesi, G. Forlani, F. Cerrelli, M. Lenzi, R. Manini, S. Natale, E. Vanni, N. Villanova, N. Melchionda and 1 more. Hepatology, 2003

2,737 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology

View Paper: https://www.semanticscholar.org/paper/5f95c6e7528f6d990f36968173c99554b3415776

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrinology, Diabetes and Metabolism

1. Z. Younossi
2. Gerald I. Shulman
3. Q. Anstee
4. M. Rinella
5. S. Klein
6. Robert D. Goldin
7. K. Cusi
8. C. Day

In Epidemiology

1. Z. Younossi
2. M. Rinella
3. Gerald I. Shulman
4. K. Cusi
5. A. Sanyal
6. Q. Anstee
7. E. Brunt
8. C. Day

Citations in the corpus

• Obesity and Nonalcoholic Fatty Liver Disease: Biochemical, Metabolic and Clinical Implications (2010, background): “93,94 In addition, bariatric surgery induced weight loss has considerable beneficial metabolic effects in the liver manifested by a decrease in (1) hepatic glucose production, (2) hepatic VLDL-triglyceride secretion rate, and (3) hepatic gene expression of factors that regulate hepatic inflammation ”
• The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome (2010, background): “The prevalence of the metabolic syndrome is 34% in American adults overall, but 53% in patients with hepatic steatosis, and even higher (88%) in those with NASH”
• Obesity, Oxidative Stress, Adipose Tissue Dysfunction, and the Associated Health Risks: Causes and Therapeutic Strategies (2015, background): “NAFLD has become an important public health issue because it is associated with a high risk of developing type 2 diabetes, cardiovascular complications, and dyslipidemia.(180) An increase in intrahepatic triglyceride content is associated with alterations in glucose, fatty acids, and lipoprotein met”
• Inflammasome-mediated dysbiosis regulates progression of NAFLD and obesity (2012, result): “These results indicate that (1) some metabolic aberrations associated with the dysbiosis of inflammasome-deficient mice can be horizontally transferred from one mouse to another,(2) the gut microbiota of inflammasomedeficient mice has a negative effect on NAFLD progression and glucose homeostasis, a”